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Additional observations suggest that it may influence cardiac electrical stability and thrombotic processes in preclinical studies

The progression from inflammation to fibrosis is primarily mediated by hepatic stellate cell (HSC) activation, which involves two core mechanisms: the Transforming Growth Factor-beta 1 (TGF-1) - Mothers Against Decapentaplegic Homolog 3 (Smad3) pathway and Platelet-Derived Growth Factor (PDGF) -driven epithelial-mesenchymal transition (EMT) 52 , Furthermore, Wnt ligands released by apoptotic bodies promote HSC proliferation through -catenin nuclear translocation 53

Taylor AA, Tsuji JS, Garry MR, et al